The Effect of Ischaemic-reperfusion on the Endogenous Fibrinolysis in Man
Primary Purpose
Ischaemic Heart Diseases
Status
Completed
Phase
Not Applicable
Locations
United Kingdom
Study Type
Interventional
Intervention
Forearm vascular study
Sponsored by
About this trial
This is an interventional basic science trial for Ischaemic Heart Diseases focused on measuring Ischaemia reperfusion, t-PA, Fibrinolysis, Endothelial function
Eligibility Criteria
Inclusion Criteria:
- Healthy males between 18-65 years of ages, non-smokers.
Exclusion Criteria:
- Any concurrent illness or chronic medical condition. Concurrent use of vasoactive medication. Smoking history.
Sites / Locations
- University of Edinburgh, 49 Little France Crescent
Arms of the Study
Arm 1
Arm 2
Arm Type
Sham Comparator
Active Comparator
Arm Label
1
2
Arm Description
no ischaemia - only sham. Blood pressure cuff inflation up till 10 mmHg on the upper arm for 20 mins.
Ischaemia 20 minutes. Blood pressure cuff will be inflated around the upper arm for 20 minutes to induce ischaemia.
Outcomes
Primary Outcome Measures
Net t-PA release from the endothelium after ischaemia reperfusion
Secondary Outcome Measures
Change in forearm blood flow after ischaemia reperfusion
Change in platelet-monocyte-binding after ischaemia reperfusion
Full Information
NCT ID
NCT00789451
First Posted
November 10, 2008
Last Updated
October 22, 2010
Sponsor
University of Edinburgh
Collaborators
University of Aarhus, University of Oxford
1. Study Identification
Unique Protocol Identification Number
NCT00789451
Brief Title
The Effect of Ischaemic-reperfusion on the Endogenous Fibrinolysis in Man
Official Title
The Effect of Ischaemic-reperfusion on the Endogenous Fibrinolysis in Man
Study Type
Interventional
2. Study Status
Record Verification Date
October 2010
Overall Recruitment Status
Completed
Study Start Date
August 2008 (undefined)
Primary Completion Date
October 2010 (Actual)
Study Completion Date
October 2010 (Actual)
3. Sponsor/Collaborators
Name of the Sponsor
University of Edinburgh
Collaborators
University of Aarhus, University of Oxford
4. Oversight
Data Monitoring Committee
No
5. Study Description
Brief Summary
Heart attacks are usually caused by a blood clot blocking an artery supplying blood to the heart. Current treatments are designed at relieving this blockage as quickly as possible to minimise damage to the heart muscle. However in restoring the supply of blood local damage known as "ischaemia-reperfusion injury" may occur. The aim of this study is to assess how clot forming and clot dissolving pathways are affected during this process, and examine the role of a natural inflammatory hormone, bradykinin. This will help us to understand the mechanism by which ischaemia-reperfusion injury may occur and to devise new treatments for heart attacks.
6. Conditions and Keywords
Primary Disease or Condition Being Studied in the Trial, or the Focus of the Study
Ischaemic Heart Diseases
Keywords
Ischaemia reperfusion, t-PA, Fibrinolysis, Endothelial function
7. Study Design
Primary Purpose
Basic Science
Study Phase
Not Applicable
Interventional Study Model
Crossover Assignment
Masking
InvestigatorOutcomes Assessor
Allocation
Randomized
Enrollment
12 (Anticipated)
8. Arms, Groups, and Interventions
Arm Title
1
Arm Type
Sham Comparator
Arm Description
no ischaemia - only sham. Blood pressure cuff inflation up till 10 mmHg on the upper arm for 20 mins.
Arm Title
2
Arm Type
Active Comparator
Arm Description
Ischaemia 20 minutes. Blood pressure cuff will be inflated around the upper arm for 20 minutes to induce ischaemia.
Intervention Type
Procedure
Intervention Name(s)
Forearm vascular study
Intervention Description
Forearm blood flow measured by venous occlusion plethysmography during interarterial infusion of substance P (2,4,8 pmol/min). Venous blood sampling via cannula in antecubital fossa.
Primary Outcome Measure Information:
Title
Net t-PA release from the endothelium after ischaemia reperfusion
Time Frame
Throughout the study
Secondary Outcome Measure Information:
Title
Change in forearm blood flow after ischaemia reperfusion
Time Frame
throughout the study
Title
Change in platelet-monocyte-binding after ischaemia reperfusion
Time Frame
Throughout the study
10. Eligibility
Sex
Male
Minimum Age & Unit of Time
18 Years
Maximum Age & Unit of Time
65 Years
Accepts Healthy Volunteers
Accepts Healthy Volunteers
Eligibility Criteria
Inclusion Criteria:
Healthy males between 18-65 years of ages, non-smokers.
Exclusion Criteria:
Any concurrent illness or chronic medical condition. Concurrent use of vasoactive medication. Smoking history.
Overall Study Officials:
First Name & Middle Initial & Last Name & Degree
David E Newby, PhD, FRCP
Organizational Affiliation
University of Edinburgh
Official's Role
Study Director
First Name & Middle Initial & Last Name & Degree
Rajesh K Kharbanda, PhD, FRCP
Organizational Affiliation
University of Oxford
Official's Role
Study Director
Facility Information:
Facility Name
University of Edinburgh, 49 Little France Crescent
City
Edinburgh
ZIP/Postal Code
EH16 4SB
Country
United Kingdom
12. IPD Sharing Statement
Citations:
PubMed Identifier
21083644
Citation
Pedersen CM, Cruden NL, Schmidt MR, Lau C, Botker HE, Kharbanda RK, Newby DE. Remote ischemic preconditioning prevents systemic platelet activation associated with ischemia-reperfusion injury in humans. J Thromb Haemost. 2011 Feb;9(2):404-7. doi: 10.1111/j.1538-7836.2010.04142.x. No abstract available.
Results Reference
derived
Learn more about this trial
The Effect of Ischaemic-reperfusion on the Endogenous Fibrinolysis in Man
We'll reach out to this number within 24 hrs